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medial preoptic area of the hypothalamus 227 medulla 225--6 orexin (hypocretin)- containing neurons 226--7 pedunculopontine tegmentum (PPT) 226 pons 225--6 pontine reticular formation 225--6 suprachiasmatic nucleus 226 thalamus 227--8 glutamate receptor agonistsantagonists clinical potential 239--40 competitive AMPA receptor antagonist 235 competitive NMDA receptor antagonist 234--5 effects on brain arousal 228--40 effects on sleep and EEG 228--40 ionotropic receptor manipulations 228--35 metabotropic receptor manipulations 235--9 noncompetitive NMDA receptor antagonists 228--34 potential use in sleep disorders 239--40 kainate 225 metabotropic NMDA 225 glycine formation 225 synthesis in the CNS 146--7 turnover in the brain 146, 147 histamine and sleep-wakefulness studies 156--66 H1 receptor agonists antagonists 162--3 H2 receptor agonists antagonists 163 H3 receptor agonists antagonists 163--5 HA administration 161--2 biochemical and molecular studies 158--60 c-Fos indication of TMN neuronal activity 159--60 early work 156--7 effects of first generation antihistamines 156 electrophysiological studies 157--8 HDC (L-histidine decarboxylase) knockout mice 160 inactivation or lesion of the TMN 165--6 manipulation of HA synthesis and degradation 161 measurement of HA release and turnover 158--9 pharmacological studies 161--6 role of the posterior hypothalamus 157 glutamate receptors AMPA 225 ionotropic 225 44, 225 catabolism 146, 147 levels in the brain 146--7 neuronal pool in the brain 146, 147 non-neuronal pool in the brain (mast cells) 146 receptors 37 role in the sleepwake cycle 37--8 synthesis 30, 36--7 43 high affinity transporter system 43 mode of action 43 neurotransmitter activity 43 role in REM muscle atonia 43 glycinergic neurons, distribution 43 hallucinations 405, 406 haloperidol 438 histamine (HA) and behavior and REM sleep 156 regulation 65--6 circadian control of synthesis and release 146--7 discovery 36 discovery of neurotransmitter activity 36, 145--6 in brain mast cells inactivation by 37 Index 469 References and Rees, D.
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